Relationship between atrophy and β‐amyloid deposition in Alzheimer disease
Identifieur interne : 007192 ( Main/Exploration ); précédent : 007191; suivant : 007193Relationship between atrophy and β‐amyloid deposition in Alzheimer disease
Auteurs : Gaël Chételat [Australie, France] ; Victor L. Villemagne [Australie] ; Pierrick Bourgeat [Australie] ; Kerryn E. Pike [Australie] ; Gareth Jones [Australie] ; David Ames [Australie] ; Kathryn A. Ellis [Australie] ; Cassandra Szoeke [Australie] ; Ralph N. Martins [Australie] ; Graeme J. O'Keefe [Australie] ; Olivier Salvado [Australie] ; Colin L. Masters [Australie] ; Christopher C. Rowe [Australie]Source :
- Annals of Neurology [ 0364-5134 ] ; 2010-03.
Descripteurs français
- Pascal (Inist)
- Wicri :
- topic : Neurologie, Médecine nucléaire.
English descriptors
- KwdEn :
- Alzheimer, Alzheimer disease, Amyloid, Amyloid deposition, Amyloid load, Amyloid plaque formation, Antiamyloid therapy, Arch neurol, Atrophy, Austin health, Australian imaging, Australian imaging biomarkers, Cerebrospinal fluid, Chetelat, Clinical group, Cluster size, Cognitive, Cognitive decline, Cognitive scores, Correlation analyses, Deposition, Disease evolution, Disease process, Elderly subjects, Global, Global measures, Global neocortical, Global neocortical compound, Gray matter, Gray matter atrophy, Gray matter volume, Healthy controls, Hippocampal, Hippocampal atrophy, Hippocampus, Imaging, Impairment, Local atrophy, Matter atrophy, Medial orbitofrontal, Melbourne, Memory complaints, Neocortical, Neocortical values, Nervous system diseases, Neuroimaging data handling, Neurol, Neurology, Neurology figure, Nuclear medicine, Pathological events, Pathological mechanisms, Pathological processes, Positron emission tomography, Posterior areas, Present study, R2adj, Regression analyses, Resonance imaging, Significant differences, Significant relationship, Soluble forms, Standardized uptake value ratio, Statistical analyses, Supplementary table.
- Teeft :
- Alzheimer, Alzheimer disease, Amyloid, Amyloid deposition, Amyloid load, Amyloid plaque formation, Antiamyloid therapy, Arch neurol, Atrophy, Austin health, Australian imaging, Australian imaging biomarkers, Cerebrospinal fluid, Chetelat, Clinical group, Cluster size, Cognitive, Cognitive decline, Cognitive scores, Correlation analyses, Deposition, Disease evolution, Disease process, Elderly subjects, Global, Global measures, Global neocortical, Global neocortical compound, Gray matter, Gray matter atrophy, Gray matter volume, Healthy controls, Hippocampal, Hippocampal atrophy, Hippocampus, Imaging, Impairment, Local atrophy, Matter atrophy, Medial orbitofrontal, Melbourne, Memory complaints, Neocortical, Neocortical values, Neuroimaging data handling, Neurol, Neurology, Neurology figure, Nuclear medicine, Pathological events, Pathological mechanisms, Pathological processes, Positron emission tomography, Posterior areas, Present study, R2adj, Regression analyses, Resonance imaging, Significant differences, Significant relationship, Soluble forms, Standardized uptake value ratio, Statistical analyses, Supplementary table.
Abstract
Elucidating the role of aggregated β‐amyloid in relation to gray matter atrophy is crucial to the understanding of the pathological mechanisms of Alzheimer disease and for the development of therapeutic trials. The present study aims to assess this relationship.
Url:
DOI: 10.1002/ana.21955
Affiliations:
- Australie, France
- Basse-Normandie, Région Normandie, Victoria (État)
- Caen, Melbourne
- Université de Melbourne
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Le document en format XML
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Masters</name><affiliation wicri:level="4"><country xml:lang="fr">Australie</country><wicri:regionArea>Mental Health Research Institute, University of Melbourne, Melbourne</wicri:regionArea><placeName><settlement type="city">Melbourne</settlement><region type="état">Victoria (État)</region><settlement type="city">Melbourne</settlement></placeName><orgName type="university">Université de Melbourne</orgName></affiliation></author><author><name sortKey="Rowe, Christopher C" sort="Rowe, Christopher C" uniqKey="Rowe C" first="Christopher C." last="Rowe">Christopher C. Rowe</name><affiliation wicri:level="1"><country xml:lang="fr">Australie</country><wicri:regionArea>Department of Nuclear Medicine and Center for PET, Austin Health, Heidelberg</wicri:regionArea><wicri:noRegion>Heidelberg</wicri:noRegion></affiliation><affiliation wicri:level="4"><country xml:lang="fr">Australie</country><wicri:regionArea>Department of Medicine, Austin Health, University of Melbourne, Heidelberg</wicri:regionArea><orgName type="university">Université de Melbourne</orgName><placeName><settlement type="city">Melbourne</settlement><region type="état">Victoria (État)</region></placeName></affiliation></author></analytic><monogr></monogr><series><title level="j" type="main">Annals of Neurology</title><title level="j" type="alt">ANNALS OF NEUROLOGY</title><idno type="ISSN">0364-5134</idno><idno type="eISSN">1531-8249</idno><imprint><biblScope unit="vol">67</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="317">317</biblScope><biblScope unit="page" to="324">324</biblScope><biblScope unit="page-count">8</biblScope><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>Hoboken</pubPlace><date type="published" when="2010-03">2010-03</date></imprint><idno type="ISSN">0364-5134</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0364-5134</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Alzheimer</term><term>Alzheimer disease</term><term>Amyloid</term><term>Amyloid deposition</term><term>Amyloid load</term><term>Amyloid plaque formation</term><term>Antiamyloid therapy</term><term>Arch neurol</term><term>Atrophy</term><term>Austin health</term><term>Australian imaging</term><term>Australian imaging biomarkers</term><term>Cerebrospinal fluid</term><term>Chetelat</term><term>Clinical group</term><term>Cluster size</term><term>Cognitive</term><term>Cognitive decline</term><term>Cognitive scores</term><term>Correlation analyses</term><term>Deposition</term><term>Disease evolution</term><term>Disease process</term><term>Elderly subjects</term><term>Global</term><term>Global measures</term><term>Global neocortical</term><term>Global neocortical compound</term><term>Gray matter</term><term>Gray matter atrophy</term><term>Gray matter volume</term><term>Healthy controls</term><term>Hippocampal</term><term>Hippocampal atrophy</term><term>Hippocampus</term><term>Imaging</term><term>Impairment</term><term>Local atrophy</term><term>Matter atrophy</term><term>Medial orbitofrontal</term><term>Melbourne</term><term>Memory complaints</term><term>Neocortical</term><term>Neocortical values</term><term>Nervous system diseases</term><term>Neuroimaging data handling</term><term>Neurol</term><term>Neurology</term><term>Neurology figure</term><term>Nuclear medicine</term><term>Pathological events</term><term>Pathological mechanisms</term><term>Pathological processes</term><term>Positron emission tomography</term><term>Posterior areas</term><term>Present study</term><term>R2adj</term><term>Regression analyses</term><term>Resonance imaging</term><term>Significant differences</term><term>Significant relationship</term><term>Soluble forms</term><term>Standardized uptake value ratio</term><term>Statistical analyses</term><term>Supplementary table</term></keywords><keywords scheme="Pascal" xml:lang="fr"><term>Amyloïde</term><term>Atrophie</term><term>Démence d'Alzheimer</term><term>Pathologie du système nerveux</term></keywords><keywords scheme="Teeft" xml:lang="en"><term>Alzheimer</term><term>Alzheimer disease</term><term>Amyloid</term><term>Amyloid deposition</term><term>Amyloid load</term><term>Amyloid plaque formation</term><term>Antiamyloid therapy</term><term>Arch neurol</term><term>Atrophy</term><term>Austin health</term><term>Australian imaging</term><term>Australian imaging biomarkers</term><term>Cerebrospinal fluid</term><term>Chetelat</term><term>Clinical group</term><term>Cluster size</term><term>Cognitive</term><term>Cognitive decline</term><term>Cognitive scores</term><term>Correlation analyses</term><term>Deposition</term><term>Disease evolution</term><term>Disease process</term><term>Elderly subjects</term><term>Global</term><term>Global measures</term><term>Global neocortical</term><term>Global neocortical compound</term><term>Gray matter</term><term>Gray matter atrophy</term><term>Gray matter volume</term><term>Healthy controls</term><term>Hippocampal</term><term>Hippocampal atrophy</term><term>Hippocampus</term><term>Imaging</term><term>Impairment</term><term>Local atrophy</term><term>Matter atrophy</term><term>Medial orbitofrontal</term><term>Melbourne</term><term>Memory complaints</term><term>Neocortical</term><term>Neocortical values</term><term>Neuroimaging data handling</term><term>Neurol</term><term>Neurology</term><term>Neurology figure</term><term>Nuclear medicine</term><term>Pathological events</term><term>Pathological mechanisms</term><term>Pathological processes</term><term>Positron emission tomography</term><term>Posterior areas</term><term>Present study</term><term>R2adj</term><term>Regression analyses</term><term>Resonance imaging</term><term>Significant differences</term><term>Significant relationship</term><term>Soluble forms</term><term>Standardized uptake value ratio</term><term>Statistical analyses</term><term>Supplementary table</term></keywords><keywords scheme="Wicri" type="topic" xml:lang="fr"><term>Neurologie</term><term>Médecine nucléaire</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract">Elucidating the role of aggregated β‐amyloid in relation to gray matter atrophy is crucial to the understanding of the pathological mechanisms of Alzheimer disease and for the development of therapeutic trials. The present study aims to assess this relationship.</div></front></TEI><affiliations><list><country><li>Australie</li><li>France</li></country><region><li>Basse-Normandie</li><li>Région Normandie</li><li>Victoria (État)</li></region><settlement><li>Caen</li><li>Melbourne</li></settlement><orgName><li>Université de Melbourne</li></orgName></list><tree><country name="Australie"><noRegion><name sortKey="Chetelat, Gael" sort="Chetelat, Gael" uniqKey="Chetelat G" first="Gaël" last="Chételat">Gaël Chételat</name></noRegion><name sortKey="Ames, David" sort="Ames, David" uniqKey="Ames D" first="David" last="Ames">David Ames</name><name sortKey="Bourgeat, Pierrick" sort="Bourgeat, Pierrick" uniqKey="Bourgeat P" first="Pierrick" last="Bourgeat">Pierrick Bourgeat</name><name sortKey="Chetelat, Gael" sort="Chetelat, Gael" uniqKey="Chetelat G" first="Gaël" last="Chételat">Gaël Chételat</name><name sortKey="Ellis, Kathryn A" sort="Ellis, Kathryn A" uniqKey="Ellis K" first="Kathryn A." last="Ellis">Kathryn A. Ellis</name><name sortKey="Jones, Gareth" sort="Jones, Gareth" uniqKey="Jones G" first="Gareth" last="Jones">Gareth Jones</name><name sortKey="Martins, Ralph N" sort="Martins, Ralph N" uniqKey="Martins R" first="Ralph N." last="Martins">Ralph N. Martins</name><name sortKey="Masters, Colin L" sort="Masters, Colin L" uniqKey="Masters C" first="Colin L." last="Masters">Colin L. Masters</name><name sortKey="O Keefe, Graeme J" sort="O Keefe, Graeme J" uniqKey="O Keefe G" first="Graeme J." last="O'Keefe">Graeme J. O'Keefe</name><name sortKey="Pike, Kerryn E" sort="Pike, Kerryn E" uniqKey="Pike K" first="Kerryn E." last="Pike">Kerryn E. Pike</name><name sortKey="Pike, Kerryn E" sort="Pike, Kerryn E" uniqKey="Pike K" first="Kerryn E." last="Pike">Kerryn E. Pike</name><name sortKey="Rowe, Christopher C" sort="Rowe, Christopher C" uniqKey="Rowe C" first="Christopher C." last="Rowe">Christopher C. Rowe</name><name sortKey="Rowe, Christopher C" sort="Rowe, Christopher C" uniqKey="Rowe C" first="Christopher C." last="Rowe">Christopher C. Rowe</name><name sortKey="Salvado, Olivier" sort="Salvado, Olivier" uniqKey="Salvado O" first="Olivier" last="Salvado">Olivier Salvado</name><name sortKey="Szoeke, Cassandra" sort="Szoeke, Cassandra" uniqKey="Szoeke C" first="Cassandra" last="Szoeke">Cassandra Szoeke</name><name sortKey="Villemagne, Victor L" sort="Villemagne, Victor L" uniqKey="Villemagne V" first="Victor L." last="Villemagne">Victor L. Villemagne</name><name sortKey="Villemagne, Victor L" sort="Villemagne, Victor L" uniqKey="Villemagne V" first="Victor L." last="Villemagne">Victor L. Villemagne</name><name sortKey="Villemagne, Victor L" sort="Villemagne, Victor L" uniqKey="Villemagne V" first="Victor L." last="Villemagne">Victor L. Villemagne</name></country><country name="France"><region name="Région Normandie"><name sortKey="Chetelat, Gael" sort="Chetelat, Gael" uniqKey="Chetelat G" first="Gaël" last="Chételat">Gaël Chételat</name></region><name sortKey="Chetelat, Gael" sort="Chetelat, Gael" uniqKey="Chetelat G" first="Gaël" last="Chételat">Gaël Chételat</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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